Once you're insulin resistant, you're on your way to getting diabetes, which is what happens when your pancreas can't keep up with the resistance and make enough insulin.

Your pancreas makes insulin to bring it back down, which is good.

The mycelium is producing enzymes peroxidases that break carbon hydrogen bonds.

Enzymes are breaking forth sugars.

That's what enzymes are doing.

And insulin resistance, as its name suggests, is when your cells get increasingly resistant to the effect of insulin trying to do its job.

So now, when insulin says to a cell,

I had become insulin resistant.

Now you've all heard of at least insulin.

This is a typical insulin injection.

More importantly, I don't have insulin resistance.

But what atrazine does is it turns on an enzyme, the machinery if you will, aromatase, that converts testosterone into estrogen.

What if obesity isn't the cause of insulin resistance at all?

I'll start with insulin, diabetes medication in the U.S.

This is a pen, it's called an insulin pen, preloaded.

What is that? It's an antiviral defense enzyme.

Now failure to produce enough insulin is incompatible with life.

We know that 30 million obese Americans in the United States don't have insulin resistance.

But insulin accelerates the conversion of calories into fat.

And we do OK. Insulin's a pretty good therapy.

He ground it up and he managed to get out a couple of chemicals one, the enzyme, he called luciferase the substrate, he called luciferin after Lucifer the Lightbearer.

And enzymes are the secret, kind of, ingredient in dough that brings out flavor.

Why couldn't we just inject the pancreas with something to regenerate the pancreas early on in the disease, perhaps even before it was symptomatic?

Adherence rates. How many people are taking their insulin every day?

Have you been keeping up a normal diet since the last shot of insulin?

We're also combining cellulases, the enzymes that break down complex sugars into simple sugars and fermentation in the same cell for producing ethanol.

This DNA copy can now be amplified using PCR.

So if you can be obese and not have insulin resistance, and you can be lean and have it, this suggests that obesity may just be a proxy for what's going on.

The daf 2 hormone receptor is very similar to the receptor for the hormone insulin and IGF 1.

If you present with a diseased pancreas we'd like to take cells from that organ.

And we call that a gal stripping technique.

I became almost maniacally obsessed in trying to understand the real relationship between obesity and insulin resistance.

Now, most researchers believe obesity is the cause of insulin resistance.

So with antithrombin in that goat that goat has been genetically modified so that the molecules of its milk actually include the molecule of antithrombin that GTC Genetics wants to create.

There's one called TOR, which is another nutrient sensor, like the insulin pathway.

The enzymes remanufactured the hydrocarbons into carbohydrates fungal sugars.

When we become insulin resistant, the homeostasis in that balance deviates from this state.

Insulin is a hormone that promotes the uptake of nutrients into your tissues after you eat a meal.

Trying to replace pancreas, trying to replace nerves that can help us with Parkinson's.

According to the source, the first autopsy gave the cause of death as pancreatitis .

So many reactions involving enzymes and proteins are sensitive to the acid content of the ocean.

It's hydrated dough we call the soaker that helps to start enzyme activity.

You have on your left an olive oil droplet, and this olive oil droplet gets attacked by enzymes.

It turned out the cell that we were trying to transplant into had a nuclease, an enzyme that chews up DNA on its surface, and was happy to eat the synthetic DNA that we gave it and never got transplantations.

And we thought it had something to do with this protein called AMP kinase, an energy protein.